November
2004
, Volume
17
, Number
11
Pages
1,242
-
1,249
Authors
Phil H.
Smith
,
1
John A.
Howie
,
2
Anthony J.
Worland
,
1
Rebecca
Stratford
,
2
and
Lesley A.
Boyd
1
Affiliations
1Department of Disease and Stress Biology, John Innes Centre, Colney Lane, Norwich Research Park, Norwich, NR4 7UH, U.K.; 2Monsanto UK Ltd., The Maris Centre, Hauxton Road, Trumpington, Cambridge, CB2 2LQ, U.K.
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RelatedArticle
Accepted 8 June 2004.
Abstract
Two mutants were isolated in wheat that showed enhanced resistance towards Puccinia striiformis f. sp. tritici, the fungal causal agent of yellow rust. The altered phenotype of I3-48 is due to a minimum of two mutation events, each showing a partial, additive effect, with one mutation segregating with a deletion on the long arm of chromosome 4D. In the case of I3-54, the enhanced resistance is due to a single, dominant mutation. In both mutants, the expression of the enhanced resistance is growth-stage specific. With I3-54, the full resistance phenotype is apparent from the third seedling leaf onwards, while with I3-48, a full resistance phenotype is only seen on the tenth and subsequent leaves. In addition to the enhanced resistance towards yellow rust, I3-48 also shows enhanced resistance towards brown rust, and I3-54 shows enhanced resistance to powdery mildew.
JnArticleKeywords
Additional keywords:
disease resistance,
suppressor genes.
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ArticleCopyright
© 2004 The American Phytopathological Society